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10.1016/j.ejca.2018.03.012 Summary Keywords mTORC1, mitochondria, mitochondrial functions, cancer, therapy Citation de la Cruz Lpez KG, Toledo Guzmn ME, Snchez EO and Garca Carranc A (2019) mTORC1 as a Regulator of Mitochondrial Functions and a Therapeutic Target in Cancer

HDAC1 governs iron homeostasis independent of histone deacetylation in iron-overload murine models

To assess potential organ toxicity, an extensive toxicity analysis was carried out

Under hypoxia, cell metabolism shifts away from mitochondrial OXPHOS to glycolysis for energy production, which is primarily mediated by activation of HIF-1 signaling

ME/CFS impacts all ages, races, and socioeconomic groups (4, 12)

L-Lysine also contributes to energy production when converted to acetyl coenzyme A, one of the principal fuels for the Krebs cycle