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Nevertheless, it remains unclear whether these microbial alterations represent primary drivers of PD pathogenesis or secondary consequences of disease-associated factorsincluding dietary modifications, pharmacological interventions (particularly levodopa, which alters gut motility and microbiota), and diminished physical activity

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Treatment with FGF21 enhances insulin sensitivity and reduces the expression of mammalian target of rapamycin complex 1 (mTORC1) within the same timeframe, demonstrating the potential interplay between FGF21 and mTORC1 to facilitate glucometabolic signaling [54]

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As a pivotal molecule in the GSH redox system, GSSG levels directly indicate cellular OS status [50, 51]