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Despite the continuous improvement of supportive treatment methods, the current clinical management of sepsis remains predominantly reactive, lacking targeted intervention strategies that can effectively interrupt the vicious cycle of organ dysfunction (9, 10)

Moreover, the increased apoptosis of type II AECs in IPF patients can directly cause alveolar collapse, thus accelerating the progression of pulmonary interstitial fibrosis (101)

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Both groups experienced improvements in physical and mood symptom scores, but group 1 had more pronounced effects

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