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For instance, a recently published article showed that the radio-resistant Pca cells strongly rely on Gln metabolism to maintain oxidative homeostasis

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PubChem, US National Library of Medicine

independently reported that N-myristylation of FSP1 is essential for its anti-ferroptotic activity [3, 4]

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Notably, ROS scavenging by N-acetylcysteine (NAC) inhibits high-glucose-induced ferroptosis, confirming that oxidative stress-mediated ferroptosis contributes to DR pathogenesis (162)