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The metabolic and mitochondrial disturbances have a two-way relationship with amyloid-beta and tau pathology, neuroinflammation, and oxidative stress, thus creating a self-sustaining cycle of neurodegeneration

Third, the prognostic role of GPX4 for LUSC was not demonstrated in this study

Further experimental studies and clinical trials are now necessary to document the relative importance of the oxidant stress caused by an altered redox potential associated with an elevation of homocysteine level, in comparison with alternative molecular mechanisms

Gastric alpha-synuclein immunoreactive inclusions in Meissners and Auerbachs plexuses in cases staged for Parkinsons disease-related brain pathology

Recent data suggest that SAM coordinately regulates homocysteine remethylation and catabolism by inhibiting methionine-conserving enzymes with low K m values and activating methionine-catabolizing enzymes with higher K m values (Finkelstein, 1990

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