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Furthermore, the primary sources of free radicals during cerebral ischaemia-reperfusion include calcium-dependent oxidative enzymes such as xanthine oxidase, NADPH oxidase, and nitric oxide synthase, as well as the degradation of membrane and mitochondrial phospholipids [14]

Low to intermediate levels of ROS and RNS are necessary for the maintenance of many important physiological functions, redox homeostasis, and the regulation of key transcription factors

These values are several orders of magnitude higher than those in the human body

2008 Jan 24 [cited 2025 Apr 19];358(4):e4

Contemporary research indicates that neoplastic cells undergoing ferroptosis may also discharge DAMPs into the extracellular milieu through autophagy, thereby enlisting and stimulating immune cells

As a result, it induces ferroptosis, a form of non-apoptotic cell death [1]