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Thus, even if the primary CFTR-dependent alteration in innate immunity is primarily an epithelial defect, there is a failure of the recruited and resident leukocytes to adequately control the hyperinflammatory state in the airway, and there is ample documentation that the presence of activated PMNs in the airway does not correlate with bacterial eradication

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Churg A, Wang RD, Tai H, et al

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