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The primary underlying pathomechanism is most likely a decreased hepatic GHR expression related to insulin deficiency, resulting in GH-resistance and impaired hepatic IGF-1 synthesis, which stimulates pituitary GH-secretion by a feedback mechanism [188, 189]

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Originally isolated in 1973, GHK-Cu has become one of the most extensively studied peptides in regenerative medicine, with natural plasma levels declining from 200 ng/ml at age 20 to 80 ng/ml by age 60

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