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Re-expression of wild-type POR, but not its mutants with defective electron transfer activity, in POR -deficient cells restored ferroptosis sensitivity, indicating that the electron transfer activity of POR is required for its function in ferroptosis regulation (Yan et al., 2020)

In practical terms, this means ketogenic strategies could serve both preventive and therapeutic roles: Pre-injury nutritional ketosis may prime the brains metabolic flexibility

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the authors of the study go on to make the following statements: Left ventricular dysfunction may result from iron deficiency The cardiomyopathy associated with iron deficiency is reversible

albicans loads across these organs in the disseminated infection model (Fig