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Long-term persistence of immune activation and proinflammation with latent and overt autoimmunity are etiological factors in clinical manifestation(s) of PASC 233

Enzymes HALY-1 and Y51H4A.7/UROC-1 are required for SKN-1 activation in AMDH-1 mutants Through the EMS suppressor screen, we also identified 3 independent alleles of haly-1 (uth92, uth93, uth95) , the conserved histidine ammonia lyase that is rate limiting for the histidine catabolism pathway, as suppressors of SKN-1 activity (fig 3a, supplemental figure 2c)

Nonetheless, it could be useful in developing biomarkers for disease and the efficacy of a treatment (200)

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