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The tryptophan metabolite 3-hydroxyanthranilic acid suppresses T cell responses by inhibiting dendritic cell activation

Nature, 408(6809), 239247

199,200,201 Although some pharmacological approaches using IGF1 have shown protective effects against cardiac injury and dysfunction in animal experimental studies, 202,203 clinical trials reported that chronic administration of IGF1 to human participants did not confer significant cardioprotective effects and may even cause cardiac fibrosis

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This interaction decreases the channels sensitivity to iron ions, restricts the efflux of molecules from mitochondria, and induces mitochondrial dysfunction

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