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In contrast, synthetic, proliferative VSMCswhich dominate pathologies such as atherosclerosisundergo a metabolic shift toward glycolysis and possess reduced antioxidant defenses, rendering them highly vulnerable to cuproptosis [154, 155]

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127 CXXC5 inhibits bone formation and osteoblast differentiation via interactions with Dvl, which blocks the signal transduction of Wnt/-catenin

Endogenously present in human plasma, its levels decline with age

The raised, dark, and hard mark from the wound becomes smoother, lighter, and more elastic