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It appears to stimulate the expression of growth factors and support the structural repair of axons and myelin sheaths, highlighting its potential as a therapeutic agent for various forms of neuropathy and nerve damage

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In addition, the proinflammatory markers, such as CD16, CCL3, IL-1 and TNF- increased in Sirt3 f/f Cx3cr1-Cre mice compared with Sirt3 f/f mice at day 3 after ICH, while the anti-inflammatory markers decreased at day7, suggesting that IF may act through microglial Sirt3 to manipulate neuroinflammation after ICH