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Remarkable enhancements were noted across all groups, with the most significant declines in HAM-D, BDI, and CGI scores occurring within the high-dose Rhodiola cohort when compared to both the low-dose and control groups

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Notably, bile acid signaling pathways can also contribute to OA pathology, as indicated by the alleviation of murine OA after suppression of farnesoid X receptor-the receptor for the bile acid glycoursodeoxycholic acid (GUDCA), which upregulates intestinal glucagon-like peptide-1 secretion (Yang et al., 2025)

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