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Chen-Roetling and Regan (2017), on the other hand, demonstrated that selective overexpression of HO-1 in astrocytes attenuates bloodbrain barrier disruption, reduces perihematoma cell injury, improves neurological deficits, and lowers mortality in an ICH model using rat autologous arterial blood

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However, PINK1 overexpression did not enhance CSE-induced mitophagy or restore the mitophagy impairment caused by PRKN knockdown, suggesting that PRKN protein levels may be the primary rate-limiting factor in PINK1/PRKN-mediated mitophagy ( Although the PINK1/Parkin pathway is generally regarded as the dominant mechanism regulating mitophagy, mitophagy can still be maintained in the absence of PINK1 or Parkin