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Description

(2024) 13:e70526

tPA and proteolysis in the neurovascular unit

Its working mechanism is allosteric facilitation of HGF/c-Met signaling: Dihexa binds HGF with high affinity (reported K d ~65 pM in the original work) and is proposed to potentiate HGF dimerization and c-Met phosphorylation at subthreshold endogenous HGF concentrations

Androgen receptor variants confer castration resistance in prostate cancer by counteracting antiandrogen-induced ferroptosis

Most adenomas were incidentally detected during IVF treatment, raising questions about the need for screening in these patients

Research interest centers on the compound's proposed capacity to potentiate HGF activity at the MET receptor, with particular attention to downstream effects on synaptic density and cognitive function-related signalling in neurological research models