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Neuroinflammation: Environmental Mechanisms and Opportunities | Austin Perlmutter, MD Neuroinflammation is increasingly recognized as a driver of both cognitive dysfunction and mood disturbance yet it remains poorly understood, under-assessed, and rarely addressed in clinical practice

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Silencing of protein kinase D2 induces glioma cell senescence via p53-dependent and -independent pathways

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Thus, this mechanism is particularly beneficial for managing T2DM, a condition characterized by impaired insulin function and hyperglycemia [156]