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We therefore postulated that IGF-1R mAbs could represent a viable therapeutic tool to target IGF-1 action, and potentially mimic the beneficial effects associated with diminished IGF-1 signaling observed in animal models

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DLPFC n = 7/14: p = 0.912)

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Pharmacology 54 , 9297 (1997)

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Pull back the plunger to your desired volume, filling the syringe with air

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Tumor suppressor PDCD4 represses internal ribosome entry site-mediated translation of antiapoptotic proteins and is regulated by S6 kinase 2

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