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Carnitine acyltransferases exist in tissue-specific isoforms with distinct kinetic characteristics and with significant modulatory targets involved in fatty acid metabolism and coenzyme-A (CoA) release [15]

Such a wide range makes the true risk hard to quantify

PLoS Comput Biol 2007

On the 4th day after the ischemia simulation, the change in the respiratory function of the mitochondria, the opening time of the mitochondrial permeability transition pore, the mitochondrial membrane potential, the concentration of intracellular calcium and the size of the cerebral necrosis zone were determined in rats brain supernatant

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These data were consistent with another study where TMAO pre-treatment for 24 h significantly reduced NO production after ATP stimulation in BAEC-1, indicating the potential involvement of TMAO in damaging the endothelial-dependent vasodilatory mechanism [57]