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Identifying at-risk individuals and treating them in a prodromal phase based on their driving pathophysiological process could prevent the manifestation of PD, or at least alter the disease progression (Ascherio and Schwarzschild, At present, two approaches could be feasible to investigate this matter: the a-priori stratification of study cohorts (based on assumed mitochondrial dysfunction) or the in-vivo assessment of mitochondrial dysfunction (by neuroimaging methods or blood biomarkers) (Prasuhn et al., 2019)

Aliment Pharmacol Ther 42:273285 Schechter I, Berger A (1967) On the size of active site in proteases

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Genetic defects in Indian Wilson disease patients and genotype-phenotype correlation

Omega-3 fatty acids: evidence basis for treatment and future research in psychiatry