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Forouzanfar et al

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234 DSF/Cu treatment also profoundly impairs mitochondrial homeostasis, increasing the free iron pool and exacerbating lipid peroxidation, ultimately culminating in ferroptosis triggered by p62 phosphorylation-mediated NRF2 accumulation

Superoxide dismutase in redox biology: the roles of superoxide and hydrogen peroxide

The residue is replaced by aspartate in poorly active ScGrx8, by histidine in protein disulfide isomerases and by proline in thioredoxins 11,12,33

NP versions of VCO or GSH, or their combination, significantly reduced tissue TNF- and IL-1 levels in rats compared to those treated with conventional drugs (Fig