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ATF1, ATF5 and ATF6 predominantly act as pro-inflammatory drivers, while ATF3 and ATF7 serve as negative feedback regulators

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Among the main obstacles, particularly concerning oral administration, are low solubility, reduced intestinal absorption, hepatic first-pass metabolism, degradation during gastrointestinal transit, interactions with the food matrix, and high individual variability, including the influence of the intestinal microbiota [50]