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Angiogenesis, wound-repair, and preclinical systemic effects In preclinical models, GHK-Cu promotes angiogenesis (new blood-vessel formation), attracts repair cells such as macrophages and capillary cells to injury sites, and accelerates wound healing across a range of tissues and species skin, hair follicles, gastrointestinal lining, bone, and more

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Similarly, ROS are similarly closely related to NETs in GBM TME

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