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Collectively, these findings indicate that astaxanthin supports synaptic plasticity through coordinated functional, structural, and molecular mechanisms, providing a mechanistic basis for its reported cognitive benefits ( 3 Biochemical and molecular mechanisms in AD Alzheimers disease is considered a neurodegenerative disorder that is progressive and mainly affects cognition, such as disrupting memory, learning, and decision-making

2 O 2 , ERO1 creates hyperoxidizing microdomains that favor oxidation of the regulatory cysteines in ERO1, and prevent its activity (Sevier et al., 2007)

Shin et al., 2005) and a reduced glutamate clearance capacity in the R6/2 mice could only be revealed by treatment with EAAT inhibitors or glutamate (Behrens et al., 2009)

That said, like any medical treatment, there are potential side effects to be aware of

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