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doi: 10.1056/NEJMoa021423

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Since 24 h treatment of adipocytes with an NNMT inhibitor increased intracellular levels of SAM and NAD + , it is likely that prolonged exposures to NNMT inhibitor during differentiation may have resulted in significantly enhanced intracellular flux of SAM and NAD +

It is important to note that the glucose-raising effect of glucagon receptor activation is largely counterbalanced by the GLP-1 and GIP components