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Cells were cultured on coverslip-bottom 35 mm dishes (MatTek P35G-1.5-14-C) until reaching approximately 70% confluency

LCA supplementation thwarted oxidative damages provoked by HFD, as supported by the decreased MitoSOX Red and DHE fluorescence signal, the reduced MDA levels and the rise of SOD levels in the serum and atria of obese mice (Figures 4AH)

46 Despite the scarcity of research on thyroid cancer cachexia, the available data revealed a strikingly high prevalence of thyroid cancer cachexia among at-risk patients, with 39.9% and even 69% in one cohort study

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Excitotoxicity: Neurotransmitter imbalance and synaptic dysfunction Excitotoxicity in MMAemia arises from glutamate/GABAglutamine metabolic reprogramming, which alters neuronal excitability and involves altered glutamatergic synapses, ion channel activity, and synaptic transmission (Denley et al