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Our clinical model (Table) achieved an NPV of 87.0% in the validation cohort

The progression from a completely normal artery to a clogged or atherosclerotic one follows a very clear path: an apoB containing particle gets past the endothelial layer into the subendothelial space, the particle and its cholesterol content is retained, immune cells arrive, an inflammatory response ensues fixing the apoB containing particles in place AND making more space for more of them

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The uncertainty around a given PocDoc result can be defined by the 95% confidence interval

By measuring TER and inulin flux, Seth et al