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Injectable L-carnitine works primarily by aiding the body in energy production, utilizing fat as a fuel source

This phenomenon is often underestimated when considering the real therapeutic effects of MCFAs in muscle tissues affected by long-chain fatty acid oxidation disorders, which seem more likely mediated by the hepatic supply of MCFA-derived ketone bodies rather than by MCFAs directly acting as a carbon source in muscle

The amount of cytochrome c oxidase negative muscle fibers in treated mice decreased by 30%, the citrate synthase activity in muscle as a marker of mitochondrial biogenesis was doubled, liver lipid levels were restored and muscle mitochondrial ultrastructure was normalized (no ragged red fiber-like muscle fibers or mitochondria with distorted structure and cristae on electron microscopy)

Characterization of TMAO productivity phenotypes by using OCCT may also help explore key human microbiome signatures of TMA synthesis from carnitine [11]

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