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GR knockout seemed to affect these pathways particularly, hinting at a metabolic recalibration in response to the redox imbalance

Patients with severe disease exhibited a larger fraction of TM-expressing classical monocytes in peripheral blood than controls (flu symptomatic patients that tested negative for SARS-CoV-2 RT-PCR)

OBM Transplant

1a, 2a, b, and Supplementary Fig

GSH is considered the main redox buffer in a cell because of the large amount of reducing equivalents supplied by GSH [9]

Unlike predictive DILI, idiosyncratic hepatotoxicity induced by certain drugs (amoxicillin clavulanate, cephalosporins, diclofenac, fluoroquinolones, isoniazid, macrolide antibiotics and nitrofurantoin) may depend on the activation of the adaptive immune system