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HOXB1 expression was induced just before the onset of MEIS1 expression and maintained until d9 of cardiac differentiation (Fig

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Inhibition of GCH1 reduced BH4 and resulted in enhanced erastin-induced iron accumulation and lipid peroxidation, thereby enhancing ferroptosis sensitivity, while BH4 supplementation suppressed ferroptosis-related changes caused by GCH1 downregulation

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