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ROS-induced mtDNA damage impairs mitochondrial OXPHOS, forcing tumor cells to rely on glycolysis for ATP production ( Figure 1 2.1 Mitochondrial metabolic regulation of immune cells T cells rely on OXPHOS and fatty acid oxidation in the resting state, but switch to aerobic glycolysis and fatty acid synthesis upon activation to support proliferation ( Figure 2 )

10.1016/j.ejps.2018.01.046 95 PerteghellaS.RassuG.GaviniE.ObinuA.BariE.MandracchiaD.et al (2021)

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It can be considered a very good option for regular methylcobalamin therapy

It should be noted that we do not include mitochondrial glutathione, which the Jaeschke group has found to be fundamental in APAP pathophysiology [36]

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