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The therapeutic vitality of these antineoplastic agents, such as daunorubicin and doxorubicin, has been compromised by increased resistance and dose-dependent toxicities including severe nephrotoxicity.87 The mechanism of anthracycline associated nephrotoxicity is not clearly understood but it has been proposed that anthracycline metabolism may induce the mitochondrial toxicities such as membrane disruption, impairment of calcium homeostasis, induction of free radical, lipid peroxidation and release of cytochrome C that may contribute toward apoptosis or necrosis of glomerular and tubular cells.8890 Montilla et al

10.3390/metabo12121158 90 JuJ.KimS

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Recognizing HU as a risk factor for CVDs, gout, and renal dysfunction underscores the importance of proactive management strategies aimed at reducing UA levels and mitigating associated complications

Trends Genet 24, 398407