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Histological analysis reveals that aged kidneys accumulate senescent cells primarily in tubular regions, identified by loss of Lamin B1 (LMNB1), elevated senescence-associated -galactosidase activity, and expression of inflammatory IL-6

Pre-clinical trials have shown favorable toxicity results, and it is considered to be safe by most researchers

Food and Drug Administration has flagged BPC-157 among bulk substances reviewed for compounding, and long-term human safety has not been established

Several stresses such as oncogene induction, oxidative environment, telomere shortening or radiation exposure are key drivers of senescence induction 1,2,3

These conditions arise from microvascular damage and vascular hyperpermeability, driven by inflammatory and pro-angiogenic factors in the eye [37]