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Prescribers should document clinical rationale and obtain informed consent that covers the absence of Phase III trial data in this specific indication

In a 2018 review published in Current Pharmaceutical Design , Sikiric et al

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A recent study reported that conditional knockout of FSP1 or GPX4 increased the sensitivity of cell ferroptosis in an IRI-AKI mouse model[101]

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Our findings, together with previous studies identifying SHIN1 as a negative modulator of psoriasiform reactions in IMQ mouse model [15], provide a strong rationale for the development of SHIN1-based topical formulations and other metabolism-targeted therapeutic strategies for psoriasis and potentially for other IL-22-driven hyperproliferative skin disorders, including NMSC [52]