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An update on the involvement of inflammatory mediators in Parkinsons disease pathogenesis

FIGURE 3 Protection Against AQ-QI- and DEAQ-QI-Induced Apoptosis by GSTP1 To test whether AQ-QI and DEAQ-QI exposure induced apoptosis and to study the protective potential of GSTP1, activity of caspase 3 was measured under normal and GSH-depleted conditions ( Figure 4 )

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Therefore, reducing intracellular GSH levels has emerged as a promising strategy to overcome therapeutic resistance and improve treatment efficacy [18]

An innovative design to establish proof of concept of the antidepressant effects of the NR2B subunit selective N-methyl-D-aspartate antagonist, CP-101,606, in patients with treatment-refractory major depressive disorder