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In addition, we also revealed transcriptomic perturbations of mitochondrial- and ROS-related pathways in Ube3a-null MEFs [30]

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In addition, we found that this is also true for the non-pathogenic Onygenaceae species Amauroascus mutatus and Byssoonygena ceratinophila

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Mitochondrial malfunction triggers ROS overproduction and an influx of Ca 2+ , resulting in vacuole formation, MAPK activation, and the initiation of paraptosis in prostate cancer cells [151]