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So if at some point you notice I removed you, that's because we never chatted and thus I don't remember who you are

Copper overload is a defining feature of Wilsons disease where mutations to the copper transporter ATP7B lead to impaired copper efflux and resultant accumulation in multiple organ systems including the CNS 39

Seeuws 2017)

Union Physiological Sci

During zinc insufficiency, its mRNA remains associated with polysomes without being translated, while the basolateral plasma membrane protein is internalized, which minimizes the secretion of body zinc from the blood into the intestinal tract [130,131]