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2012;7(5): e38317

Serine-derived glycine together with cysteine and glutamate constitute the glutathione molecule, essential for the maintenance of the redox state allowing cellular metabolic functioning and chemoresistance

Hepatology (2013) 57:33950

His articles help interpret glutathione as a cellular redox entity while keeping antioxidant terminology tied to literature context

This process is morphologically marked by small mitochondria with condensed membrane densities, and biochemically by iron overload and GSH depletion, leading to loss of antioxidant defense and irreversible oxidative damage ( 3.2 Key molecular drivers of ferroptosis Iron Overload: Excess intracellular ferrous iron (Fe 2+ ) catalyzes the Fenton reaction, producing hydroxyl radicals (OH) that initiate lipid peroxidation cascades

doi: 10.1021/acs.jproteome.0c00853 51 DemichevVMessnerCBVernardisSILilleyKSRalserM