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Mammalian cells are thought to maintain an appropriate pool of cytosolic iron by balancing iron uptake and storage in ferritin cages with the release of ferritin-sequestered iron through degradation of these proteinaceous cages (Arosio et al, 2009)

Administration of IL-33 induces airway hyperresponsiveness and goblet cell hyperplasia in the lungs in the absence of adaptive immune system

The salient features cohesive to this thesis have illuminated the following: the augmentation of NO secretion via increase in expression and/or activity of eNOS