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Figure 1 illustrates how diet interfaces with the multifactorial pathophysiology of AD, highlighting anatomical vulnerability, molecular cascades, and potential dietary interventions

4.1.3 Targeting ferroptosis to improve tubular damage in DN Ferroptosis mediates tubular injury in DN, suggesting that inhibiting tubular injury ferroptosis may provide a therapeutic approach for DN

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These results suggest that inhibiting depalmitoylation of ATG2 can mimic the consequences observed upon loss of ATG2, leading to impaired autophagosome formation (Velikkakath et al, 2012)

Since the inhibitory effect of phytate on zinc absorption is concentration-dependent, the molar phytate: zinc-ratio of the diet (Table 1) is applied to estimate zinc bioavailability [49] and was shown to be more important than the phytate content of the product itself [171,172]

NAD metabolism: Implications in aging and longevity