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The induction of ICD by CUR/miR155@DssD-Hb NPs may stimulate the maturation of DCs in vitro via the release of "eat me" signals and tumor-associated antigens

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Moreover, the use of NET inhibitors currently focus more on animal experiments, and in-depth research is lacking in clinical trials

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Subsequent follow-up research also confirmed that deletion of the shared TLRs signaling 135 or adaptor including MyD88, TRAM, and TRIF leads to a reduction in plaque burden to varying degrees

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