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CER is able to regulate iron metabolism levels in HCC cells to modulate iron toxicity, and when protein levels are reduced, It leads to increased intracellular iron levels and accumulation of lipid peroxides, Then promotes erastin and RSL3-induced iron toxicity, which may be achieved by relying on FPN, The inhibitory effect of CER on ferroptosis was abolished when FPN expression was inhibited by shRNA [154]

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First, the expression of glucose transporter 1 (GLUT1) is upregulated, increasing glucose uptake to enhance subsequent aerobic glycolysis [26]

Pollen mixing may dilute the potentially toxic compounds originating from toxic plants (58)

Chapter 11 - Fluorescence-based analysis of trafficking in mammalian cells