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Overproduction of reactive oxygen species (ROS) in the nucleus pulposus cells (NPCs) is a critical contributor to the progression of IDD through increasing oxidative stress in the microenvironment of the extracellular matrix (ECM) 5 , leading to ferroptosis and long-term persistent inflammation 6,7 , which would cause the loss of structure and function in the intervertebral disc (IVD) ultimately

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[DOI] [PubMed] [Google Scholar] 63.Mahmoud A.M., Hozayen W.G., Ramadan S.M

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