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fragilis can release a unique pro-inflammatory LPS-subtype and fragilysin, leading to an impaired intestinal paracellular and transcellular epithelial barrier, thereby allowing an already leaky barrier a gateway for the enhanced entry of microbiome-derived neuro- and entero-toxins into the circulation (Lukiw et al., 2021)

In light of these changes, identifying region-specific regulatory mechanisms of mitophagy is crucial for intervening in the aging process

doi: 10.1016/S0092-8674(04)00400-3 60 WilkinsonDJPiaseckiMAthertonPJ

Trionfini and Benigni 2017)

Analyte abundance was correlated with questionnaire items to find associations between potential markers of disease and disease manifestations

Targeting GPER1 to suppress autophagy as a male-specific therapeutic strategy for iron-induced striatal injury