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Thus, as the SLC1A5 variant is the only currently known glutamine transporter in the mitochondrial inner membrane 16 , targeting the SLC1A5 variant could be an effective strategy for selectively inhibiting glutamine metabolism in cancer cells (Fig

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in 1972 established that the catalytic activity of GPx depended on selenium status of the erythrocytes [3]

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While the exact cause of AD onset is still unclear, the prevailing hypothesis is that the increase in amyloid- (A) plaques and neurofibrillary tangles (NFTs) in the brain leads to synaptic dysfunction, and thus the onset of AD (Dias et al., 2014