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doi: 10.1111/cts.12993 46 Kryczyk-KozioJProchownikEBaewska-GruszczykASowiaczekMSunQSchomburgLet al

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To begin to evaluate potential targeting of c-MYC in endometriosis therapy, we utilized the well-characterized human endometriotic epithelial cell line, 12Z which expressed eGFP (Figure 2 depicts representative immunofluorescence for Omomyc localization with white arrows highlighting nuclear expression of Dox-induced Omomyc expression

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S-Acetyl-L-glutathione (50 M) increases intracellular GSH levels in primary fibroblasts derived from patients with glutathione synthetase deficiency.1 It induces apoptosis in Daudi, Raji, and Jurkat lymphoma cells when used at a concentration of 5 mM