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Indeed, studies on genetically modified mice have shown that deficiencies in NK cells, CD8+ cytotoxic lymphocytes, or CD4+ helper T cells all increase susceptibility to carcinogen-induced cancer (Teng 2008

Kelly, N., Friend, K., Boyle, P., Zhang, X

However, as reviewed in ref

Cancer stem-like properties and gefitinib resistance are dependent on purine synthetic metabolism mediated by the mitochondrial enzyme MTHFD2

On the other hand, acquired resistance emerges during treatment as a result of an evolutionary process in which cancer cells adapt to survive therapeutic pressures