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The association between microevolutionary capacity and propensity to cause IMD in several hypervirulent lineages is particularly intriguing, as it has been proposed that meningococcal virulence results from the accidental emergence of invasive variants during carriage

KEAP1 protein has also been shown to co-localise with intracellular inclusions in MNs of postmortem ALS spinal cord [245], possibly through an interaction with p62 observed in several ALS inclusions [246]

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In its absence, Teff cells become sensitive to conjugated BAs (CBAs), leading to oxidative stress and elevated production of pro-inflammatory cytokines like IFN- and TNF-