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Based on the presumed mechanisms that trigger these effects, it can be assumed that no genotoxic effects occur at doses below certain limits, although lower thresholds are possible if the glutathione reserve is reduced

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At the same time, hepcidin levels increase, thereby accelerating the degradation of FPN, closing the iron transport outlet to the blood, and reducing the transport of iron from small intestinal epithelial cells and macrophages to the blood ( In addition, ferroptosis is closely related to iron metabolism under iron overload

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