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JUN upregulation drives aberrant transposable element mobilization, associated innate immune response, and impaired neurogenesis in Alzheimer's disease

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If the substrate is deglutathionylated, the first stage proceeds by the monothiol mechanism, but the glutathionylated intermediate product Grx-SSG then releases GSH yielding the intramolecular disulfide bond Grx(S 2 ) between the catalytic cysteine residue and one of the recycling cysteines

Furthermore, in vitro studies using RAW264.7 cells indicate that HG reduces OCs autophagy, thus amplifying OCs formation [187]

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